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  Indian J Med Microbiol
 

Figure 2: Histopathological examination of liver sections from (a) normal rats showing normal hepatic architecture, (b) thioacetamide-intoxicated rats showing severe disturbances in hepatic architecture and portal tract inflammation with severe lymphocyte infiltration and focal spotty necrosis (black arrow), (c) silymarin-treated rats showing almost normal hepatic architecture with mild dilation of sinusoids and lymphocyte infiltration, and (d) resveratrol-treated rats showing normal hepatic architecture and mild number of chronic inflammatory cells infiltration (H and E, ×200)

Figure 2: Histopathological examination of liver sections from (a) normal rats showing normal hepatic architecture, (b) thioacetamide-intoxicated rats showing severe disturbances in hepatic architecture and portal tract inflammation with severe lymphocyte infiltration and focal spotty necrosis (black arrow), (c) silymarin-treated rats showing almost normal hepatic architecture with mild dilation of sinusoids and lymphocyte infiltration, and (d) resveratrol-treated rats showing normal hepatic architecture and mild number of chronic inflammatory cells infiltration (H and E, ×200)